HPV18 oncoproteins driven expression of PKM2 reprograms HeLa cell metabolism to maintain aerobic glycolysis and viability
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Virology
Link
https://link.springer.com/content/pdf/10.1007/s13337-022-00776-w.pdf
Reference62 articles.
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3. Carretero J, et al. Dysfunctional AMPK activity, signalling through mTOR and survival in response to energetic stress in LKB1-deficient lung cancer. Oncogene. 2007;26(11):1616–25.
4. Clower CV, et al. The alternative splicing repressors hnRNP A1/A2 and PTB influence pyruvate kinase isoform expression and cell metabolism. Proc Natl Acad Sci U S A. 2010;107(5):1894–9.
5. Cortes-Cros M, et al. M2 isoform of pyruvate kinase is dispensable for tumor maintenance and growth. Proc Natl Acad Sci U S A. 2013;110(2):489–94.
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1. PKM2 induces mitophagy through the AMPK-mTOR pathway promoting CSFV proliferation;Journal of Virology;2024-03-19
2. Human papillomavirus associated cervical lesion: pathogenesis and therapeutic interventions;MedComm;2023-09-14
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