7-Aminobutyric Acid (GABA) Removal from the Synaptic Cleft: A Postsynaptic Event?

Author:

Cupello Aroldo,Hydén Holger

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Cellular and Molecular Neuroscience,General Medicine

Reference44 articles.

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2. Ayala, G. F., and Johnston, D. (1980). Electrophysiological studies in simple neuronal systems. InAntiepileptic Drugs: Mechanisms of Action (Glaser, G. H., Penry, J. K., and Woodbury, D. M., Eds.), Raven Press, New York, pp. 339–352.

3. Barker, J. L., and Mather, D. A. (1981). GABA analogues activate channels of different duration on cultured mouse spinal neurones.Science 212358–361.

4. Barker, J. L., and Segal, M. (1983). Coexistence of transmitter functions in excitable membranes of cultured CNS neurons. InCoexistence of Neuroactive Substances in Neurons (Chan Palay, V., and Palay, S. L., Eds.), John Wiley and Sons, New York, pp. 339–362.

5. Constanti, A., Krnjevic, K., and Nistri, A. (1979). Intracellular injections of GABA have a hyperpolarizing effect on motoneurones.J. Physiol. (London)29360–61P.

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