Heat shock protein 27 in the pathogenesis of COVID-19 and non-COVID acute respiratory distress syndrome-Reference-Cited by-同舟云学术

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Heat shock protein 27 in the pathogenesis of COVID-19 and non-COVID acute respiratory distress syndrome

Published:2023-11 Issue:6 Volume:28 Page:877-887
ISSN:1355-8145
Container-title:Cell Stress and Chaperones
language:en
Short-container-title:Cell Stress and Chaperones

Author:

Chiu Michael H.^ORCID,Gershkovich Benjamin,Yu Ian-Ling,O’Brien Edward R.,Deng Jingti,McDonald Braedon

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Link

https://link.springer.com/content/pdf/10.1007/s12192-023-01381-6.pdf

Reference33 articles.

1. The cellular "networking" of mammalian Hsp27 and its functions in the control of protein folding, redox state and apoptosis;Arrigo;Adv Exp Med Biol,2007

2. COVID-19 versus non-COVID-19 acute respiratory distress syndrome: comparison of demographics, physiologic parameters, inflammatory biomarkers, and clinical outcomes;Bain;Ann Am Thorac Soc,2021

3. Batulan Z, Pulakazhi Venu VK, Li Y et al (2016) Extracellular release and signaling by heat shock protein 27: role in modifying vascular inflammation. Front Immunol 7:285

4. Elevated HSP27 levels during attacks in patients with multiple sclerosis;Ce;Acta Neurol Scand,2011

5. HSP25 Vaccination attenuates atherogenesis via upregulation of LDLR expression, lowering of PCSK9 levels and curbing of inflammation;Chen;Arterioscler Thromb Vasc Biol,2021

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