Chlamydia trachomatis L2/434/Bu Favors Hypoxia for its Growth in Human Lymphoid Jurkat Cells While Maintaining Production of Proinflammatory Cytokines
Author:
Funder
KAKENHI
Publisher
Springer Science and Business Media LLC
Subject
Applied Microbiology and Biotechnology,General Medicine,Microbiology
Link
https://link.springer.com/content/pdf/10.1007/s00284-022-02961-y.pdf
Reference39 articles.
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2. Fischer SF, Vier J, Kirschnek S, Klos A, Hess S, Ying S, Häcker G (2004) Chlamydia inhibit host cell apoptosis by degradation of proapoptotic BH3-only proteins. J Exp Med 200:905–916. https://doi.org/10.1084/jem.20040402
3. McLean CA, Stoner BP, Workowski KA (2007) Treatment of lymphogranuloma venereum. Clin Infect Dis 44(3):S147–S152. https://doi.org/10.1086/511427
4. Sellami H, Said-Sadier N, Znazen A, Gdoura R, Ojcius DM, Hammami A (2014) Chlamydia trachomatis infection increases the expression of inflammatory tumorigenic cytokines and chemokines as well as components of the toll-like receptor and NF-κB pathways in human prostate epithelial cells. Mol Cell Probes 28:147–154. https://doi.org/10.1016/j.mcp.2014.01.006
5. Eltzschig HK, Carmeliet P (2011) Hypoxia and inflammation. N Engl J Med 364:656–665. https://doi.org/10.1056/NEJMra0910283
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