Partial defect in hepatic glutathione S-transferase activity in a case of Rotor’s syndrome

Author:

Adachi Yukihiko,Yamamoto Toshio

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology

Reference17 articles.

1. Rotor AB, et al: Familial non-hemolytic jaundice with direct van den Bergh reaction. Acta Med Phil 5: 37, 1948

2. Schiff L, et al: Familial non-hemolytic jaundice with conjugated bilirubin in the serum. N Engl J Med 260: 1315, 1959

3. Wolpert E, et al: Abnormal sulfobromophthalein metabolism in Rotor’s syndrome and obligate heterozygotes. N Engl J Med 296: 1099, 1977

4. Kawasaki H, et al: Dyel clearance studies in Rotor’s syndrome. Am J Gastroenterol 71: 380, 1979

5. Knoflach P, et al: Benigne chronische konjugierte Hyperbilirubinämie: Rotor-Syndrome oder hepatischer Speicherdefekt? Z Gastroenterol 17: 32, 1979

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2. 6. Progress in the Studies of Jaundice - Including the Implication of Drug Metabolism;Nihon Naika Gakkai Zasshi;2007

3. Hepatic Bilirubin Metabolism: Physiology and Pathophysiology;Liver and Environmental Xenobiotics;1997

4. Genetic heterogeneity of the human glutathione transfereses: A complex of gene families;Pharmacology & Therapeutics;1990-01

5. 10.2957/kanzo.30.1040;Kanzo;1989

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