Voiding Dysfunction in Multiple Sclerosis: Disease Review and Management
Author:
Publisher
Springer Science and Business Media LLC
Subject
Molecular Biology,Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/s11884-013-0213-8.pdf
Reference80 articles.
1. Noonan CW, Kathman SJ, White MC. Prevalence estimates for MS in the United States and evidence of an increasing trend for women. Neurology. 2002;58:136–8.
2. Weiner. Multiple sclerosis is an inflammatory T-cell-mediated autoimmune disease. Arch Neurol. 2004;61:1613–5.
3. Bansil S, Cook SD. C R-K. Multiple sclerosis: immune mechanism and update on current therapies. Ann Neurol. 1995;37:S87–S101.
4. Lucchinetti C, Bruck W, Parisi J, et al. Heterogeneity of multiple sclerosis lesions: implications for the pathogeneis of demyelination. Ann Neurol. 2000;47:707–17.
5. Trapp BD, Peterson J, Ransohoff RM, et al. Axonal transection in the lesions of multiple sclerosis. N Engl J Med. 1998;338:278–85.
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