Comparison of ion channel inhibitor combinations for limiting secondary degeneration following partial optic nerve transection
Author:
Funder
National Health and Medical Research Council
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://link.springer.com/article/10.1007/s00221-018-5414-0/fulltext.html
Reference52 articles.
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2. Anderson A (2002) Mechanisms and pathways of inflammatory responses in CNS trauma: spinal cord injury. J Spinal Cord Med 25:70–79
3. Annunziato L, Amoroso S, Pannaccione A et al (2003) Apoptosis induced in neuronal cells by oxidative stress: role played by caspases and intracellular calcium ions. Toxicol Lett 139:125–133. https://doi.org/10.1016/S0378-4274(02)00427-7
4. Arancibia-Carcamo I, Attwell D (2014) The node of Ranvier in CNS pathology. Acta Neuropathol 128:161–175. https://doi.org/10.1007/s00401-014-1305-z
5. Banik NL, McAlhaney WW, Hogan EL (1985) Calcium-stimulated proteolysis in myelin: evidence for a Ca2+-activated neutral proteinase associated with purified myelin of rat CNS. J Neurochem 45:581–588. https://doi.org/10.1111/j.1471-4159.1985.tb04026.x
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1. Secondary Degeneration Impairs Myelin Ultrastructural Development in Adulthood following Adolescent Neurotrauma in the Rat Optic Nerve;International Journal of Molecular Sciences;2023-02-07
2. Acute Cellular and Functional Changes With a Combinatorial Treatment of Ion Channel Inhibitors Following Spinal Cord Injury;Frontiers in Molecular Neuroscience;2020-06-25
3. Damage Mechanisms to Oligodendrocytes and White Matter in Central Nervous System Injury: The Australian Context;Journal of Neurotrauma;2020-03-01
4. Comparing modes of delivery of a combination of ion channel inhibitors for limiting secondary degeneration following partial optic nerve transection;Scientific Reports;2019-10-25
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