Cigarette smoke-induced malignant transformation via STAT3 signalling in pulmonary epithelial cells in a lung-on-a-chip model-Reference-Cited by-同舟云学术

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Cigarette smoke-induced malignant transformation via STAT3 signalling in pulmonary epithelial cells in a lung-on-a-chip model

Published:2020-08-17 Issue:4 Volume:3 Page:383-395
ISSN:2096-5524
Container-title:Bio-Design and Manufacturing
language:en
Short-container-title:Bio-des. Manuf.

Author:

Hou Wei,Hu Siyi,Yong Ken-tye,Zhang Jie^ORCID,Ma Hanbin

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Industrial and Manufacturing Engineering,Materials Science (miscellaneous),Biomedical Engineering,Biotechnology

Link

https://link.springer.com/content/pdf/10.1007/s42242-020-00092-6.pdf

Reference41 articles.

1. Hogg JC, Timens W (2009) The pathology of chronic obstructive pulmonary disease. Annu Rev Pathol 4:435–459. https://doi.org/10.1146/annurev.pathol.4.110807.092145

2. Milara J, Peiró T, Serrano A, Cortijo J (2013) Epithelial to mesenchymal transition is increased in patients with COPD and induced by cigarette smoke. Thorax 68(5):410–420. https://doi.org/10.1136/thoraxjnl2012-201761

3. Decramer M, Janssens W, Miravitlles M (2012) Chronic obstructive pulmonary disease. Lancet 379(9823):1341–1351. https://doi.org/10.1016/s0140-6736(11)60968-9

4. Alwis KU, de Castro BR, Morrow JC, Blount BC (2015) Acrolein exposure in U.S. tobacco smokers and non-tobacco users: NHANES 2005–2006. Environ Health Perspect 123(12):1302–1308. https://doi.org/10.1289/ehp.1409251

5. Portillo-Lara R, Annabi N (2016) Microengineered cancer-on-a-chip platforms to study the metastatic microenvironment. Lab Chip 16(21):4063–4081. https://doi.org/10.1039/c6lc00718j

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