Sustained increase in rat myocardial α1A-adrenoceptors induced by 6-hydroxydopamine treatment involves a decelerated receptor turnover
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/BF00261437.pdf
Reference26 articles.
1. Badia A, Sallés J, Gascón S (1994) Acute 6-hydroxydopamine treatment specifically up-regulates rat myocardial α1A- adrenoceptors. Can J Physiol Pharmacol 72:327
2. Barturen F, Garciá-Sevilla JA (1992) Long-term treatment with desipramine increases the turnover of α2-adrenoceptors in the rat brain. Mol Pharmacol 42:846–855
3. Benfey MD (1990) Function of myocardial α-adrenoceptors. Life Sci 46:743–757
4. Bradford MM (1976) A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal Biochem 72:248–254
5. Chess-Williams RG, Broadley KJ, Sheridan DJ (1987) Cardiac postjunctional α1- and β-adrenoceptors: effects of chronic chemical sympathectomy with 6-hydroxydopamine. J Recept Res 7:713–728
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