The Roles of Matrix Stiffness and ß-Catenin Signaling in Endothelial-to-Mesenchymal Transition of Aortic Valve Endothelial Cells
Author:
Funder
Canadian Institutes of Health Research
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Biomedical Engineering
Link
http://link.springer.com/article/10.1007/s13239-018-0363-0/fulltext.html
Reference35 articles.
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2. Calve, S., and H. G. Simon. Extracellular control of limb regeneration. Dordrecht: Springer, pp. 257–266, 2010.
3. Calve, S., and H.-G. Simon. Biochemical and mechanical environment cooperatively regulate skeletal muscle regeneration. FASEB J. 26:2538–2545, 2012.
4. Chen, J. H., W. L. K. Chen, K. L. Sider, C. Y. Y. Yip, and C. A. Simmons. β-Catenin mediates mechanically regulated, transforming growth factor-β1-induced myofibroblast differentiation of aortic valve interstitial cells. Arterioscler. Thromb. Vasc. Biol. 31:590–597, 2011.
5. Chen, J. H., and C. A. Simmons. Cell-matrix interactions in the pathobiology of calcific aortic valve disease: critical roles for matricellular, matricrine, and matrix mechanics cues. Circ. Res. 108:1510–1524, 2011.
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