Cell Cycle Withdrawal Limit the Regenerative Potential of Neonatal Cardiomyocytes
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Biomedical Engineering
Link
https://link.springer.com/content/pdf/10.1007/s13239-021-00551-w.pdf
Reference22 articles.
1. Aurora, A. B., E. R. Porrello, W. Tan, A. I. Mahmoud, J. A. Hill, R. Bassel-Duby, et al. Macrophages are required for neonatal heart regeneration. J. Clin. Investig. 124(3):1382–1392, 2014. https://doi.org/10.1172/JCI72181.
2. Bergmann, O., S. Zdunek, A. Felker, M. Salehpour, K. Alkass, S. Bernard, et al. Dynamics of cell generation and turnover in the human heart. Cell 161(7):1566–1575, 2015. https://doi.org/10.1016/j.cell.2015.05.026.
3. Blom, J. N., X. Lu, P. Arnold, and Q. Feng. Myocardial infarction in neonatal mice, a model of cardiac regeneration. J. Vis. Exp., 2016. https://doi.org/10.3791/54100.
4. Cullere, X., E. Plovie, P. M. Bennett, C. A. MacRae, and T. N. Mayadas. The cerebral cavernous malformation proteins CCM2L and CCM2 prevent the activation of the MAP kinase MEKK3. Proc. Natl Acad. Sci. U S A 112(46):14284–14289, 2015. https://doi.org/10.1073/pnas.1510495112.
5. Drenckhahn, J. D., Q. P. Schwarz, S. Gray, A. Laskowski, H. Kiriazis, Z. Ming, et al. Compensatory growth of healthy cardiac cells in the presence of diseased cells restores tissue homeostasis during heart development. Dev. Cell 15(4):521–533, 2008. https://doi.org/10.1016/j.devcel.2008.09.005.
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