Nupr1 deletion protects against glucose intolerance by increasing beta cell mass-Reference-Cited by-同舟云学术

Nupr1 deletion protects against glucose intolerance by increasing beta cell mass

Published:2013-07-31 Issue:11 Volume:56 Page:2477-2486
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Barbosa-Sampaio Helena C.,Liu Bo,Drynda Robert,Rodriguez de Ledesma Ana M.,King Aileen J.,Bowe James E.,Malicet Cédric,Iovanna Juan L.,Jones Peter M.,Persaud Shanta J.,Muller Dany S.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00125-013-3006-x.pdf

Reference42 articles.

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2. Vasseur S, Folch-Puy E, Hlouschek V et al (2004) p8 improves pancreatic response to acute pancreatitis by enhancing the expression of the anti-inflammatory protein pancreatitis-associated protein I. J Biol Chem 279:7199–7207

3. Jiang YF, Vaccaro MI, Fiedler F, Calvo EL, Iovanna JL (1999) Lipopolysaccharides induce p8 mRNA expression in vivo and in vitro. Biochem Biophys Res Commun 260:686–690

4. Vasseur S, Hoffmeister A, Garcia-Montero A et al (2003) Mice with targeted disruption of p8 gene show increased sensitivity to lipopolysaccharide and DNA microarray analysis of livers reveals an aberrant gene expression response. BMC Gastroenterol 3:25

5. Encinar JA, Mallo GV, Mizyrycki C et al (2001) Human p8 is a HMG-I/Y-like protein with DNA binding activity enhanced by phosphorylation. J Biol Chem 276:2742–2751

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