The CD19 signalling molecule is elevated in NOD mice and controls type 1 diabetes development-Reference-Cited by-同舟云学术

The CD19 signalling molecule is elevated in NOD mice and controls type 1 diabetes development

Published:2013-09-08 Issue:12 Volume:56 Page:2659-2668
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Ziegler Alexandra I.,Le Page Melanie A.,Maxwell Mhairi J.,Stolp Jessica,Guo Haoyao,Jayasimhan Abhirup,Hibbs Margaret L.,Santamaria Pere,Miller Jacques F.,Plebanski Magdalena,Silveira Pablo A.,Slattery Robyn M.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00125-013-3038-2.pdf

Reference50 articles.

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2. Hamilton-Williams EE, Palmer SE, Charlton B, Slattery RM (2003) Beta cell MHC class I is a late requirement for diabetes. Proc Natl Acad Sci U S A 100:6688–6693

3. Serreze DV, Gaskins HR, Leiter EH (1993) Defects in the differentiation and function of antigen presenting cells in NOD/Lt mice. J Immunol 150:2534–2543

4. Jun HS, Santamaria P, Lim HW, Zhang ML, Yoon JW (1999) Absolute requirement of macrophages for the development and activation of beta-cell cytotoxic CD8+ T cells in T cell receptor transgenic NOD mice. Diabetes 48:34–42

5. Weaver DJ Jr, Poligone B, Bui T, Abdel-Motal UM, Baldwin AS Jr, Tisch R (2001) Dendritic cells from nonobese diabetic mice exhibit a defect in NF-kappa B regulation due to a hyperactive I kappa B kinase. J Immunol 167:1461–1468

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