Glucolipotoxicity promotes the capacity of the glycerolipid/NEFA cycle supporting the secretory response of pancreatic beta cells-Reference-Cited by-同舟云学术

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Glucolipotoxicity promotes the capacity of the glycerolipid/NEFA cycle supporting the secretory response of pancreatic beta cells

Published:2022-01-12 Issue:4 Volume:65 Page:705-720
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Oberhauser Lucie,Jiménez-Sánchez Cecilia,Madsen Jesper Grud Skat,Duhamel Dominique,Mandrup Susanne,Brun Thierry,Maechler Pierre^ORCID

Funder

European Foundation for the Study of Diabetes

AGLA 2018 Swiss Lipid Research Award

Fondation pour la Recherche sur le Diabète

Swiss Commission for Technology and Innovation

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00125-021-05633-x.pdf

Reference49 articles.

1. Maechler P (2002) Mitochondria as the conductor of metabolic signals for insulin exocytosis in pancreatic beta-cells. Cell Mol Life Sci 59:1803–1818. https://doi.org/10.1007/PL00012507

2. Nolan CJ, Madiraju MS, Delghingaro-Augusto V, Peyot ML, Prentki M (2006) Fatty acid signaling in the beta-cell and insulin secretion. Diabetes 55(Suppl 2):S16–S23

3. Corkey BE, Glennon MC, Chen KS, Deeney JT, Matschinsky FM, Prentki M (1989) A role for malonyl-CoA in glucose-stimulated insulin secretion from clonal pancreatic beta-cells. J Biol Chem 264:21608–21612. https://doi.org/10.1016/S0021-9258(20)88227-1

4. Brun T, Roche E, Kim KH, Prentki M (1993) Glucose regulates acetyl-CoA carboxylase gene expression in a pancreatic beta-cell line (INS-1). J Biol Chem 268:18905–18911. https://doi.org/10.1016/S0021-9258(17)46712-3

5. Prentki M, Peyot ML, Masiello P, Madiraju SRM (2020) Nutrient-induced metabolic stress, adaptation, detoxification, and toxicity in the pancreatic β-cell. Diabetes 69:279–290. https://doi.org/10.2337/dbi19-0014

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