Non-esterified fatty acids are deleterious for human pancreatic islet function at physiological glucose concentration-Reference-Cited by-同舟云学术

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Non-esterified fatty acids are deleterious for human pancreatic islet function at physiological glucose concentration

Published:2004-03 Issue:3 Volume:47 Page:463-469
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Dubois M.,Kerr-Conte J.,Gmyr V.,Bouckenooghe T.,Muharram G.,D’Herbomez M.,Martin-Ponthieu A.,Vantyghem M. C.,Vandewalle B.,Pattou F.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00125-004-1347-1.pdf

Reference41 articles.

1. Kahn SE (2000) The importance of the beta-cell in the pathogenesis of type 2 diabetes mellitus. Am J Med 108 [Suppl 6a]:2S–8S

2. Eizirik DL, Korbutt GS, Hellerstrom C (1992) Prolonged exposure of human pancreatic islets to high glucose concentrations in vitro impairs the beta-cell function. J Clin Invest 90:1263–1268

3. Marshak S, Leibowitz G, Bertuzzi et al. (1999) Impaired b-cell functions induced by chronic exposure of cultured human pancreatic islets to high glucose. Diabetes 48:1230–1236

4. Lee Y, Hirose H, Ohneda M, Johnson JH, McGarry JD, Unger RH (1994) Beta-cell lipotoxicity in the pathogenesis of non-insulin-dependent diabetes mellitus of obese rats: impairment in adipocyte-beta-cell relationships. Proc Natl Acad Sci USA 91:10878–10882

5. Lee Y, Hirose H, Zhou YT, Esser V, McGarry JD, Unger RH (1997) Increased lipogenic capacity of the islets of obese rats: a role in the pathogenesis of NIDDM. Diabetes 46:408–413

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