Biphasic dynamics of beta cell mass in a mouse model of congenital hyperinsulinism: implications for type 2 diabetes-Reference-Cited by-同舟云学术

Biphasic dynamics of beta cell mass in a mouse model of congenital hyperinsulinism: implications for type 2 diabetes

Published:2021-02-09 Issue:5 Volume:64 Page:1133-1143
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Tornovsky-Babeay Sharona,Weinberg-Corem Noa,Ben-Haroush Schyr Rachel^ORCID,Avrahami Dana^ORCID,Lavi Judith,Feleke Eseye,Kaestner Klaus H.^ORCID,Dor Yuval^ORCID,Glaser Benjamin^ORCID

Funder

The Alex U Soyka pancreatic cancer fund

European Foundation for the Study of Diabetes

Waldholtz / Pakula family

National Institute of Diabetes and Digestive and Kidney Diseases

Israel Science Foundation

Robert M. and Marilyn Sternberg Family Charitable Foundation

British Israel Research Exchange

Ernest and Bonnie Beutler Research Program of Excellence in Genomic Medicine

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00125-021-05390-x.pdf

Reference43 articles.

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2. Fonseca SG, Gromada J, Urano F (2011) Endoplasmic reticulum stress and pancreatic beta-cell death. Trends Endocrinol Metab 22(7):266–274. https://doi.org/10.1016/j.tem.2011.02.008

3. Kaneto H, Kajimoto Y, Fujitani Y et al (1999) Oxidative stress induces p21 expression in pancreatic islet cells: Possible implication in beta-cell dysfunction. Diabetologia 42(9):1093–1097. https://doi.org/10.1007/s001250051276

4. Robertson R, Zhou H, Zhang T, Harmon JS (2007) Chronic oxidative stress as a mechanism for glucose toxicity of the beta cell in type 2 diabetes. Cell Biochem Biophys 48(2–3):139–146. https://doi.org/10.1007/s12013-007-0026-5

5. Robertson RP, Harmon J, Tran PO, Tanaka Y, Takahashi H (2003) Glucose toxicity in beta-cells: Type 2 diabetes, good radicals gone bad, and the glutathione connection. Diabetes 52(3):581–587. https://doi.org/10.2337/diabetes.52.3.581

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