Transcriptional profiling of myotubes from patients with type 2 diabetes: no evidence for a primary defect in oxidative phosphorylation genes-Reference-Cited by-同舟云学术

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Transcriptional profiling of myotubes from patients with type 2 diabetes: no evidence for a primary defect in oxidative phosphorylation genes

Published:2008-08-22 Issue:11 Volume:51 Page:2068-2077
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Frederiksen C. M.,Højlund K.,Hansen L.,Oakeley E. J.,Hemmings B.,Abdallah B. M.,Brusgaard K.,Beck-Nielsen H.,Gaster M.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00125-008-1122-9.pdf

Reference44 articles.

1. Højlund K, Beck-Nielsen H (2006) Impaired glycogen synthase activity and mitochondrial dysfunction in skeletal muscle. Markers or mediators of insulin resistance in type 2 diabetes. Curr Diabetes Rev 2:375–395

2. Højlund K, Staehr P, Hansen BF et al (2003) Increased phosphorylation of skeletal muscle glycogen synthase at NH2-terminal sites during physiological hyperinsulinemia in type 2 diabetes. Diabetes 52:1393–1402

3. Gaster M, Ottosen PD, Vach W et al (2003) GLUT4 expression in human muscle fibres is not correlated with intracellular triglyceride (TG) content. Is TG a maker or a marker of insulin resistance. APMIS 111:338–348

4. Levin K, Daa Schroeder H, Alford FP et al (2001) Morphometric documentation of abnormal intramyocellular fat storage and reduced glycogen in obese patients with type II diabetes. Diabetologia 44:824–833

5. Beck-Nielsen H, Vaag A, Poulsen P et al (2003) Metabolic and genetic influence on glucose metabolism in type 2 diabetic subjects—experiences from relatives and twin studies. Best Pract Res Clin Endocrinol Metab 17:445–467

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