No REST for healthy beta cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00125-008-1043-7.pdf
Reference24 articles.
1. Martin D, Allagnat F, Chaffard G et al (2008) Functional significance of REST target genes in pancreatic beta cells. Diabetologia DOI 10.1007/s00125-008-0984-1
2. Atouf F, Czernichow P, Scharfmann R (1997) Expression of neuronal traits in pancreatic beta cells. Implication of neuron-restrictive silencing factor/repressor element silencing transcription factor, a neuron-restrictive silencer. J Biol Chem 272:1929–1934
3. Chong JA, Tapia-Ramirez J, Kim S et al (1995) REST: a mammalian silencer protein that restricts sodium channel gene expression to neurons. Cell 80:949–957
4. Thiel G, Hohl M (2006) RE-1 silencing transcription factor (REST)—regulation of neuronal gene expression via modification of the chromatin structure. In: Thiel G (ed) Transcription factors in the nervous system—development, brain function and disease. Wiley, Germany, pp 113–128
5. Ballas N, Grunseich C, Lu DD, Speh JC, Mandel G (2005) REST and its corepressors mediate plasticity of neuronal gene chromatin throughout neurogenesis. Cell 121:645–657
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1. Aberrant Expression of Rest4 Gene in Low-Functioning Pancreatic Beta Cell Line;Hormone and Metabolic Research;2023-02-09
2. Loss of RE-1 silencing transcription factor accelerates exocrine damage from pancreatic injury;Cell Death & Disease;2020-02
3. Disallowed and Allowed Gene Expression: Two Faces of Mature Islet Beta Cells;Annual Review of Nutrition;2016-07-17
4. Serotonin competence of mouse beta cells during pregnancy;Diabetologia;2016-04-07
5. REST represses a subset of the pancreatic endocrine differentiation program;Developmental Biology;2015-09
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