NADPH oxidase inhibition prevents beta cell dysfunction induced by prolonged elevation of oleate in rodents-Reference-Cited by-同舟云学术

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NADPH oxidase inhibition prevents beta cell dysfunction induced by prolonged elevation of oleate in rodents

Published:2013-02-21 Issue:5 Volume:56 Page:1078-1087
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Koulajian K.,Desai T.,Liu G. C.,Ivovic A.,Patterson J. N.,Tang C.,El-Benna J.,Joseph J. W.,Scholey J. W.,Giacca A.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/article/10.1007/s00125-013-2858-4/fulltext.html

Reference49 articles.

1. Giacca A, Xiao C, Oprescu AI, Carpentier AC, Lewis GF (2011) Lipid-induced pancreatic beta-cell dysfunction: focus on in vivo studies. Am J Physiol Endocrinol Metab 300:E255–E262

2. Moore PC, Ugas MA, Hagman DK, Parazzoli SD, Poitout V (2004) Evidence against the involvement of oxidative stress in fatty acid inhibition of insulin secretion. Diabetes 53:2610–2616

3. Morgan D, Oliveira-Emilio HR, Keane D et al (2007) Glucose, palmitate and pro-inflammatory cytokines modulate production and activity of a phagocyte-like NADPH oxidase in rat pancreatic islets and a clonal beta cell line. Diabetologia 50:359–369

4. Syed I, Jayaram B, Subasinghe W, Kowluru A (2010) Tiam1/Rac1 signaling pathway mediates palmitate-induced, ceramide-sensitive generation of superoxides and lipid peroxides and the loss of mitochondrial membrane potential in pancreatic beta-cells. Biochem Pharmacol 80:874–883

5. Carlsson C, Borg LA, Welsh N (1999) Sodium palmitate induces partial mitochondrial uncoupling and reactive oxygen species in rat pancreatic islets in vitro. Endocrinology 140:3422–3428

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