A label-free MALDI TOF MS-based method for studying the kinetics and inhibitor screening of the Alzheimer’s disease drug target β-secretase-Reference-Cited by-同舟云学术

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A label-free MALDI TOF MS-based method for studying the kinetics and inhibitor screening of the Alzheimer’s disease drug target β-secretase

Published:2018-09-15 Issue:28 Volume:410 Page:7441-7448
ISSN:1618-2642
Container-title:Analytical and Bioanalytical Chemistry
language:en
Short-container-title:Anal Bioanal Chem

Author:

Machálková Markéta,Schejbal Jan,Glatz Zdeněk,Preisler Jan

Funder

Ministry of Education, Youth and Sports of the Czech Republic

Grantová Agentura České Republiky

Grant Agency of Masaryk University

Publisher

Springer Science and Business Media LLC

Subject

Biochemistry,Analytical Chemistry

Link

http://link.springer.com/article/10.1007/s00216-018-1354-6/fulltext.html

Reference30 articles.

1. Dementia. World Health Organization. http://www.who.int/mediacentre/factsheets/fs362/en/ . 2017. Accessed 2 March 2018.

2. Glenner G, Wong C. Alzheimer’s disease: initial report of the purification and characterization of a novel cerebrovascular amyloid protein. Biochem Biophys Res Commun. 1984;120(3):885–90. https://doi.org/10.1016/S0006-291X(84)80190-4 .

3. Lacor PN, Buniel MC, Furlow PW, Sanz Clemente A, Velasco PT, Wood M, et al. Aβ oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer’s disease. J Neurosci. 2007;27(4):796–807. https://doi.org/10.1523/jneurosci.3501-06.2007.

4. Luo Y, Bolon B, Kahn S, Bennett BD, Babu-Khan S, Denis P, et al. Mice deficient in BACE1, the Alzheimer’s β-secretase, have normal phenotype and abolished β-amyloid generation. Nat Neurosci. 2001;4:231.

5. Cole SL, Vassar R. The Alzheimer’s disease β-secretase enzyme, BACE1. Mol Neurodegener. 2007;2(1):22.

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