Very long-chain fatty acids in diagnosis, pathogenesis, and therapy of peroxisomal disorders
Author:
Publisher
Wiley
Subject
Cell Biology,Organic Chemistry,Biochemistry
Reference18 articles.
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2. Moser, A.B., Rasmussen, M., Naidu, S., Watkins, P.A., McGuinness, M., Hajra, A.K., Chen, G., Raymond, G., Liu, A., Gordon, D., Garnaas, K., Walton, D.S., Skjeldal, O.H., Guggenheim, M.A., Jackson, L.G., Elias, E.R., and Moser, H.W. (1995) Phenotype of Patients with Peroxisomal Disorders Subdivided into 16 Complementation Groups,J. Pediatr. 127, 13–22.
3. Mosser, J., Lutz, Y., Stoeckel, M.E., Sarde, C.O., Kretz, C., Douar, A.M., Lopez, J., Aubourg, P., and Mandel, J.L. (1994) The Gene Responsible for Adrenoleukodystrophy Encodes a Peroxismal Membrane Protein,Hum. Mol. Genet. 3, 265–271.
4. Powers, J.M., Liu, Y., Moser, A., and Moser, H. (1992) The Inflammatory Myelinopathy of Adreno-Leukodystrophy: Cells, Effector Molecules, and Pathogenetic Implications,J. Neuropathol. Exp. Neurol. 51, 630–643.
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