Prostaglandin synthetase inhibitors are not beneficial in the hypercatabolic state of acute uremia in rats

Author:

Weipert J.,Peter G.,Heidland A.

Publisher

Springer Science and Business Media LLC

Subject

General Medicine

Reference18 articles.

1. Baracos V, Rodemann HP, Dinarello CA, Goldberg AL (1983) Stimulation of muscle protein degradation and prostaglandin E2 release by leukocytic pyrogen (interleukin-1). N Engl J Med 308:553–558

2. Butler RR, Jr, Wise WC, Haluska PV, Cook JA (1983) Gentamicin and indomethacin in the treatment of septic shock effects on prostacyclin and thromboxane A2 production. J Pharmacol Exp Ther 225:94–101

3. Flügel-Link RM, Salusky JB, Jones MR, Kopple JD (1983) Protein and amino acid metabolism in posterior hemicorpus of acutely uremic rats. Am J Physiol 244 (Endocrinol Metab 7):E 615-E 623

4. Haverberg L, Deckelbaum L, Bilmazes C, Munro H, Young V (1975) Myofibrillar protein turnover and urinary Nτ methylhistidine output. Biochem J 152:503–510

5. Heidland A, Hörl WH (1984) Contribution of proteases to hypercatabolism in acute renal failure. In: Robinson RR (ed) Nephrology. Springer, Berlin Heidelberg New York, pp 763–775

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