Interaction between polygalacturonase-inhibiting protein and jasmonic acid during defense activation in tomato against Botrytis cinerea
Author:
Publisher
Springer Science and Business Media LLC
Subject
Horticulture,Plant Science,Agronomy and Crop Science
Link
http://link.springer.com/content/pdf/10.1007/s10658-010-9684-z.pdf
Reference26 articles.
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2. Bishop, P. D., Makus, D. J., Pearce, G., & Ryan, C. A. (1981). Proteinase inhibitor-inducing factor activity in tomato leaves resides in oligosaccharides enzymically released from cell walls. Proceedings of the National Academy of Sciences of the United States of America, 78, 3536–3540.
3. Cervone, F., Hahn, M. G., De Lorenzo, G., Darvill, A., & Albersheim P. (1989). Host-pathogen interactions XXXIII. A plant protein converts a fungal pathogenesis factor into an elicitor of plant defense responses. Plant Physiology, 90, 542–548.
4. Diaz, J., ten Have, A., & van Kan, J. A. L. (2002). The role of ethylene and wound signaling in resistance of tomato to Botrytis cinerea. Plant Physiology, 129, 1341–1351.
5. Ding, C. K., Wang, C. Y., Gross, K. C., & Smith, D. L. (2002). Jasmonate and salicylate induce the expression of pathogenesis-related-protein genes and increase resistance to chilling injury in tomato fruit. Planta, 214, 895–901.
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