Destructive pulmonary staphylococcal infection in a boy with hyper-IgE syndrome: a novel mutation in the signal transducer and activator of transcription 3 (STAT3) gene (p.Y657S)-Reference-Cited by-同舟云学术

Destructive pulmonary staphylococcal infection in a boy with hyper-IgE syndrome: a novel mutation in the signal transducer and activator of transcription 3 (STAT3) gene (p.Y657S)

Published:2010-11-24 Issue:5 Volume:170 Page:661-666
ISSN:0340-6199
Container-title:European Journal of Pediatrics
language:en
Short-container-title:Eur J Pediatr

Author:

Liu Jin-yan,Li Qiang,Chen Ting-ting,Guo Xia,Ge Jiao,Yuan Li-xing

Publisher

Springer Science and Business Media LLC

Subject

Pediatrics, Perinatology, and Child Health

Link

http://link.springer.com/content/pdf/10.1007/s00431-010-1349-6.pdf

Reference21 articles.

1. Buckley RH, Wray BB, Belmaker EZ (1972) Extreme hyperimmunoglobulinemia E and undue susceptibility to infection. Pediatrics 49:59–70

2. Davis SD, Schaller J, Wedgwood RJ (1966) Job's syndrome. Recurrent, “cold”, staphylococcal abscesses. Lancet 1:1013–1015

3. Engelhardt KR, McGhee S, Winkler S et al (2009) Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome. Allergy Clin Immunol 124:1289–1302.e4

4. Fanconi S, Seger RA, Willi U et al (1984) Oral chloramphenicol therapy for multiple liver abscesses in hyperimmunoglobulinemia E syndrome. Eur J Pediatr 142:292–295

5. Grimbacher B, Schäffer AA, Holland SM et al (1999) Genetic linkage of hyper-IgE syndrome to chrosome 4. Am J Hum Genet 65:735–744

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