Tumour genesis syndrome: severe hypophosphatemia and hypokalemia may be ominous presenting findings in childhood acute myeloid leukaemia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology, and Child Health
Link
http://link.springer.com/article/10.1007/s00431-017-2899-7/fulltext.html
Reference15 articles.
1. Brautbar N, Leibovici H, Massry SG (1983) On the mechanism of hypophosphatemia during acute hyperventilation: evidence for increased muscle glycolysis. Miner Electrolyte Metab 9:45–50
2. Dalal BI, Brigden ML (2009) Factitious biochemical measurements resulting from hematologic conditions. Am J Clin Pathol 131:195–204
3. Filippatos TD, Milionis HK, Elisaf MS (2005) Alternations in electrolyte equilibrium in patients with acute leukemia. Eur J Haematol 75:449–460
4. Liamis G, Elisaf M (2000) Hypokalemia, hypophosphatemia and hypouricemia due to proximal renal tubular dysfunction in acute myeloid leukemia. Eur J Haematol 64:277–278
5. Majhail NS, Lichtin AE (2004) Acute leukemia with a very high leukocyte count: confronting a medical emergency. Cleve Clin J Med 71(8):633–637
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Risk factors and development of a predictive score model for tumor lysis syndrome in childhood leukemia: a 10-year experience from a single tertiary hospital in Thailand;Leukemia & Lymphoma;2024-08-18
2. Biochemical and Hematologic Profiles in B-Cell Acute Lymphoblastic Leukemia Children;Journal of Pediatric Hematology/Oncology;2023-07-14
3. Tumor Genesis Syndrome Presenting as Severe Hypophosphatemia in a Patient With T-Cell Acute Lymphoblastic Leukemia;Cureus;2023-05-10
4. Erratum to: Tumour genesis syndrome: severe hypophosphatemia and hypokalemia may be ominous presenting findings in childhood acute myeloid leukaemia;European Journal of Pediatrics;2017-07-03
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