Distortion product otoacoustic emissions in term infants after hypoxia-ischaemia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology and Child Health
Link
http://link.springer.com/content/pdf/10.1007/s00431-004-1569-8.pdf
Reference22 articles.
1. Borg E (1997) Perinatal asphyxia, hypoxia, ischemia and hearing loss. An overview. Scand Audiol 26: 77?91
2. Frolenkov GI, Belyantseva IA, Kurc M, Mastroianni MA, Kacha RB (1998) Cochlear outer hair cell electromotility can provide force for both low and high intensity distortion product otoacoustic emissions. Hear Res 126: 67?74
3. Gaskill SA, Brown AM (1993) Comparison of the level of the acoustic distortion product, 2f1?f2, with behavioural threshold audiograms from normal-hearing and hearing-impaired ears. Br J Audiol 27: 397?407
4. Harris FP, Glattke Tl (1992) The use of suppression to determine the characteristics of otoacoustic emissions. Semin Hear 13: 67
5. Jiang ZD, Brosi DM, Wang J, Xu X, Chen GQ, Shao XM, Wilkinson AR (2003) Time course of brainstem pathophysiology during first month in term infants after perinatal asphyxia, revealed by MLS BAER latencies and intervals. Pediatr Res 54: 680?687
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