Hemodynamic profile of arpromidine and its F2-substituted derivatives in comparison to impromidine in congestive heart failure
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology (medical),Toxicology,Pharmacology,Immunology
Link
http://link.springer.com/content/pdf/10.1007/BF01997365.pdf
Reference8 articles.
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2. H. R. Bristow, R. Ginsburg, W. B. S. Minobe, R. S. Cybicciotti, W. S. Sageman, K. Lurie, M. E. Billingham, D. G. Harrison and E. B. Stinson,Decreased catecholamine sensitivity and β-adrenergic receptor density in failing human hearts. N. Engl. J. Med.307, 205–211 (1982).
3. G. Baumann, S. Felix, C. D. Heideke, G. Riess, U. Loher, L. Ludwig and H. Blömer,Apparent superiority of H2-receptor-stimulation and simultaneous β-blockade over conventional treatment with β-sympathomimetic drugs in postacute myocardial infarction: cardiac effects of impromidine-a new specific H2-receptor agonist in the surviving catecholamine-insensitive myocardium. Agents and Actions15, 216–228 (1984).
4. G. Baumann, B. Permanetter and A. Wirztfeld,Possible value of H2-receptor agonists for treatment of catecholamine-insensitive congestive heart failure. Pharmacol. Ther.24, 165–172 (1984).
5. A. Buschauer,Synthesis and in-vitro pharmacology of arpromidine and related phenyl (pyridylalkyl) guanidines, a potential new class of positive inotropic drugs. J. Med. Chem.32, 1963–1970 (1989).
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