An assessment of gene-by-gene interactions as a tool to unfold missing heritability in dyslexia-Reference-Cited by-同舟云学术

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An assessment of gene-by-gene interactions as a tool to unfold missing heritability in dyslexia

Published:2015-04-28 Issue:7 Volume:134 Page:749-760
ISSN:0340-6717
Container-title:Human Genetics
language:en
Short-container-title:Hum Genet

Author:

Mascheretti S.,Bureau A.,Trezzi V.,Giorda R.,Marino C.

Funder

research fellowship from the Fonds de recherche du Québec – Santé

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics

Link

http://link.springer.com/content/pdf/10.1007/s00439-015-1555-4.pdf

Reference84 articles.

1. Akashi K, Kakizaki T, Kamiya H et al (2009) NMDA receptor GluN2B (GluR epsilon 2/NR2B) subunit is crucial for channel function, postsynaptic macromolecular organization, and actin cytoskeleton at hippocampal CA3 synapses. J Neurosci 29:10869–10882

2. American Psychiatric Association (1994) Diagnostic and statistical manual of mental disorders, 4th edn. American Psychiatry Press, Washington, DC

3. Baddeley A (2003) Working memory: looking back and looking forward. Nat Rev Neurosci 4:829–839

4. Boyle AP, Hong EL, Hariharan M et al (2012) Annotation of functional variation in personal genomes using RegulomeDB. Genome Res 22:1790–1797

5. Che A, Girgenti MJ, Loturco J (2014) The dyslexia-associated gene Dcdc2 is required for spike-timing precision in mouse neocortex. Biol Psychiatry 76:387–396

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