Effects of 2-(2-Chlorophenyl)ethylbiguanide on ERAD Component Expression in HT-29 Cells Under a Serum- and Glucose-Deprived Condition-Reference-Cited by-同舟云学术

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Effects of 2-(2-Chlorophenyl)ethylbiguanide on ERAD Component Expression in HT-29 Cells Under a Serum- and Glucose-Deprived Condition

Published:2019-02-16 Issue:4 Volume:188 Page:1009-1021
ISSN:0273-2289
Container-title:Applied Biochemistry and Biotechnology
language:en
Short-container-title:Appl Biochem Biotechnol

Author:

Oh-hashi Kentaro,Matsumoto Shiori,Sakai Takayuki,Hirata Yoko,Okuda Kensuke,Nagasawa Hideko

Funder

Grant-in-Aid for Challenging Exploratory Research

the OGAWA Science and Technology Foundation

the Koshiyama Science and Technology Foundation

Publisher

Springer Science and Business Media LLC

Subject

Molecular Biology,Applied Microbiology and Biotechnology,Biochemistry,General Medicine,Bioengineering,Biotechnology

Link

http://link.springer.com/article/10.1007/s12010-019-02969-4/fulltext.html

Reference46 articles.

1. Helenius, A., Marquardt, T., & Braakman, I. (1992). The endoplasmic reticulum as protein-folding compartment. Trends in Cell Biology, 2(8), 227–231.

2. Zhu, C., Johansen, F. E., & Prywes, R. (1997). Interaction of ATF6 and serum response factor. Molecular and Cellular Biology, 17(9), 4957–4966.

3. Calfon, M., Zeng, H., Urano, F., Till, J. H., Hubbard, S. R., Harding, H. P., Clark, S. G., & Ron, D. (2002). IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA. Nature, 415(6867), 92–96.

4. Harding, H. P., Zhang, Y., & Ron, D. (1999). Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase. Nature, 397(6716), 271–274.

5. Kim, I., Xu, W., & Reed, J. C. (2008). Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities. Nature Reviews Drug Discovery, 7(12), 1013–1030.

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1. Development of antitumor biguanides targeting energy metabolism and stress responses in the tumor microenvironment;Scientific Reports;2021-03-01

2. Suspension State Promotes Drug Resistance of Breast Tumor Cells by Inducing ABCC3 Overexpression;Applied Biochemistry and Biotechnology;2019-08-01

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