Clinical implications and mechanism of complement C1q in polymyositis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Molecular Biology,Applied Microbiology and Biotechnology,Biochemistry,General Medicine,Bioengineering,Biotechnology
Link
https://link.springer.com/content/pdf/10.1007/s12010-023-04692-7.pdf
Reference27 articles.
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2. Csorba, K., Schirmbeck, L., Dubler, D., et al. (2021). Anti-C1q autoantibodies: standard quantification and innovative ELISA. Methods in Molecular Biology, 2227, 107–114.
3. Du, Q., Huang, Y., Wang, T., et al. (2016). Porcine circovirus type 2 activates PI3K/Akt and p38 MAPK pathways to promote interleukin-10 production in macrophages via Cap interaction of gC1qR. Oncotarget, 7, 17492–17507.
4. Findlay, A. R., Goyal, N. A., & Mozaffar, T. (2015). An overview of polymyositis and dermatomyositis. Muscle & Nerve, 51, 638–656.
5. Ghebrehiwet, B., Geisbrecht, B. V., Xu, X., et al. (2019). The C1q Receptors: Focus on gC1qR/p33 (C1qBP, p32, HABP-1)(1). Seminars in Immunology, 45, 101338.
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