“Insulin-like” effects of palmitate compromise insulin signalling in hypothalamic neurons
Author:
Funder
Marsden Fund
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,Animal Science and Zoology,Biochemistry,Ecology, Evolution, Behavior and Systematics,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s00360-019-01220-0.pdf
Reference40 articles.
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2. Belsham DD, Fick LJ, Dalvi PS, Centeno ML, Chalmers JA, Lee PK, Wang Y, Drucker DJ, Koletar MM (2009) Ciliary neurotrophic factor recruitment of glucagon-like peptide-1 mediates neurogenesis, allowing immortalization of adult murine hypothalamic neurons. FASEB Journal 23(12):4256–4265. https://doi.org/10.1096/fj.09-133454
3. Benzler J, Ganjam GK, Legler K, Stohr S, Kruger M, Steger J, Tups A (2013) Acute inhibition of central c-Jun N-terminal kinase restores hypothalamic insulin signalling and alleviates glucose intolerance in diabetic mice. J Neuroendocrinol 25(5):446–454. https://doi.org/10.1111/jne.12018
4. Benzler J, Ganjam GK, Pretz D, Oelkrug R, Koch CE, Legler K, Stohr S, Culmsee C, Williams LM, Tups A (2015) Central inhibition of IKKbeta/NF-kappaB signaling attenuates high-fat diet-induced obesity and glucose intolerance. Diabetes 64(6):2015–2027. https://doi.org/10.2337/db14-0093
5. Capel F, Acquaviva C, Pitois E, Laillet B, Rigaudiere JP, Jouve C, Pouyet C, Gladine C, Comte B, Vianey Saban C, Morio B (2015) DHA at nutritional doses restores insulin sensitivity in skeletal muscle by preventing lipotoxicity and inflammation. J Nutr Biochem 26(9):949–959. https://doi.org/10.1016/j.jnutbio.2015.04.003
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