Study Protocol for the Pleiotropic Effects of Sodium–Glucose Cotransporter 2 Inhibitor on Organ-Specific Sympathetic Nerve Activity and Insulin Sensitivity in Participants with Type 2 Diabetes
Author:
Funder
Japan Association for Diabetes Education and Care
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://link.springer.com/content/pdf/10.1007/s13300-023-01497-z.pdf
Reference38 articles.
1. Thaung HPA, Baldi JC, Wang H-Y, et al. Increased efferent cardiac sympathetic nerve activity and defective intrinsic heart rate regulation in type 2 diabetes. Diabetes. 2015;64(8):2944–56. https://doi.org/10.2337/db14-0955.
2. Minatoguchi S. Heart failure and its treatment from the perspective of sympathetic nerve activity. J Cardiol. 2022;79(6):691–7. https://doi.org/10.1016/j.jjcc.2021.11.016.
3. Grassi G, Seravalle G, Dell’Oro R, Mancia G. Sympathetic mechanisms, organ damage, and antihypertensive treatment. Curr Hypertens Rep. 2011;13(4):303–8. https://doi.org/10.1007/s11906-011-0200-4.
4. Emerging Risk Factors Collaboration. Diabetes mellitus, fasting glucose, and risk of cause-specific death. N Engl J Med. 2011;364(9):829–41. https://doi.org/10.1056/NEJMoa1008862.
5. Limberg JK, Smith JA, Soares RN, et al. Sympathetically mediated increases in cardiac output, not restraint of peripheral vasodilation, contribute to blood pressure maintenance during hyperinsulinemia. Am J Physiol Circ Physiol. 2020;319(1):H162-70. https://doi.org/10.1152/ajpheart.00250.2020.
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