Urotensin II can Induce Skeletal Muscle Atrophy Associated with Upregulating Ubiquitin–Proteasome System and Inhibiting the Differentiation of Satellite Cells in CRF Mice-Reference-Cited by-同舟云学术

Urotensin II can Induce Skeletal Muscle Atrophy Associated with Upregulating Ubiquitin–Proteasome System and Inhibiting the Differentiation of Satellite Cells in CRF Mice

Published:2023-03-09 Issue:5 Volume:112 Page:603-612
ISSN:1432-0827
Container-title:Calcified Tissue International
language:en
Short-container-title:Calcif Tissue Int

Author:

Pan Yajing,Zhou Ting,Dong Xingtong,Wu Leiyun,Wang Peiwen,Wang Shiyuan,Zhang Aihua^ORCID

Funder

National Natural Science Foundation of China

Ministry of Education & National Natural Science Foundation of Beijing

Xuanwu Hospital Huizhi talent leader training program

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology,Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://link.springer.com/content/pdf/10.1007/s00223-023-01073-4.pdf

Reference28 articles.

1. Wang XH, Mitch WE (2014) Mechanisms of muscle wasting in chronic kidney disease. Nat Rev Nephrol 10(9):504–516. https://doi.org/10.1038/nrneph.2014.112

2. Changchien CY, Lin YH, Cheng YC, Chang HH, Peng YS, Chen Y (2019) Indoxyl sulfate induces myotube atrophy by ROS-ERK and JNK-MAFbx cascades. Chem Biol Interact 304:43–51. https://doi.org/10.1016/j.cbi.2019.02.023

3. Rom O, Reznick AZ (2016) The role of E3 ubiquitin-ligases MuRF-1 and MAFbx in loss of skeletal muscle mass. Free Radic Biol Med 98:218–230. https://doi.org/10.1016/j.freeradbiomed.2015.12.031

4. Foletta VC, White LJ, Larsen AE, Leger B, Russell AP (2011) The role and regulation of MAFbx/atrogin-1 and MuRF1 in skeletal muscle atrophy. Pflugers Arch 461(3):325–335. https://doi.org/10.1007/s00424-010-0919-9

5. Ravani P, Tripepi G, Pecchini P, Mallamaci F, Malberti F, Zoccali C (2008) Urotensin II is an inverse predictor of death and fatal cardiovascular events in chronic kidney disease. Kidney Int 73(1):95–101. https://doi.org/10.1038/sj.ki.5002565