Chronic type-I diabetes could not impede the anti-inflammatory and anti-apoptotic effects of combined postconditioning with ischemia and cyclosporine A in myocardial reperfusion injury-Reference-Cited by-同舟云学术

Chronic type-I diabetes could not impede the anti-inflammatory and anti-apoptotic effects of combined postconditioning with ischemia and cyclosporine A in myocardial reperfusion injury

Published:2016-10-22 Issue:1 Volume:73 Page:111-120
ISSN:1138-7548
Container-title:Journal of Physiology and Biochemistry
language:en
Short-container-title:J Physiol Biochem

Author:

Badalzadeh Reza,Azimi Ako,Alihemmati Alireza,Yousefi Bahman

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Biochemistry,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s13105-016-0530-4.pdf

Reference32 articles.

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2. Argaud L, Gateau-Roesch O, Raisky O, Loufouat J, Robert D, Ovize M (2005) Postconditioning inhibits mitochondrial permeability transition. Circulation 111:194–197

3. Badalzadeh R, Mohammadi M, Najafi M, Ahmadiasl N, Farajnia S, Ebrahimi H (2012) The additive effects of ischemic postconditioning and cyclosporine-A on nitric oxide activity and functions of diabetic myocardium injured by ischemia/reperfusion. J Cardiovasc Pharmacol Ther 17:181–189

4. Badalzadeh R, Yousefi B, Majidinia M, Ebrahimi H (2014a) Anti-arrhythmic effect of diosgenin in reperfusion-induced myocardial injury in a rat model: activation of nitric oxide system and mitochondrial KATP channel. J Physiol Sci 64:393–400

5. Badalzadeh R, Yousefi B, Tajaddini A, Ahmadian N (2014b) Diosgenin-induced protection against myocardial ischaemia-reperfusion injury is mediated by mitochondrial KATP channels in a rat model. Perfusion 0267659114566064.

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