The senescence-accelerated mouse prone-8 (SAM-P8) oxidative stress is associated with upregulation of renal NADPH oxidase system-Reference-Cited by-同舟云学术

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The senescence-accelerated mouse prone-8 (SAM-P8) oxidative stress is associated with upregulation of renal NADPH oxidase system

Published:2013-07-11 Issue:4 Volume:69 Page:927-935
ISSN:1138-7548
Container-title:Journal of Physiology and Biochemistry
language:en
Short-container-title:J Physiol Biochem

Author:

Baltanás Ana,Solesio Maria E.,Zalba Guillermo,Galindo María F.,Fortuño Ana,Jordán Joaquín

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Biochemistry,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s13105-013-0271-6.pdf

Reference36 articles.

1. Alvarez-Garcia O, Vega-Naredo I, Sierra V, Caballero B, Tomas-Zapico C, Camins A, Garcia JJ, Pallas M, Coto-Montes A (2006) Elevated oxidative stress in the brain of senescence-accelerated mice at 5 months of age. Biogerontology 7:43–52

2. Balaban RS, Nemoto S, Finkel T (2005) Mitochondria, oxidants, and aging. Cell 120:483–495

3. Baltanás A, Miguel-Carrasco JL, San José G, Cebrián C, Dotor J, Moreno MD, Borrás-Cuesta F, López B, Gonzalez A, Díez J, Fortuño A, Zalba G (2013) A synthetic peptide from transforming growth factor-beta1 type III receptor inhibits NADPH oxidase and prevents oxidative stress in the kidney of spontaneously hypertensive rats. Antioxid Redox Signal (in press)

4. Bedard K, Krause KH (2007) The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology. Physiol Rev 87:245–313

5. Bokoch GM, Knaus UG (2003) NADPH oxidases: not just for leukocytes anymore! Trends Biochem Sci 28:502–508

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