Ontogenetic quinpirole treatments fail to prime for D2 agonist-enhancement of locomotor activity in 6-hydroxydopamine-lesioned rats

Author:

Brus Ryszard,Kostrzewa Richard M.,Nowak Preemyslaw,Perry Ken W.,Kostrzewa John P.

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Reference32 articles.

1. Breese GR, AA Baumeister, TJ McCown, SG Emerick, GD Frye, K Crotty and RA Mueller (1984) Behavioural differences between neonatal and adult 6-hydroxydopamine-treated rats to dopamine agonists: relevance to neurological symptoms in clinical syndromes with reduced brain dopamine.J Pharm. Exp. Ther. 231, 343–354.

2. Breese GR, AA Baumeister, TC Napier, GD Frye and RA Mueller (1985a) Evidence that D-1 dopamine receptors contribute to the supersensitive behavioral responses induced by L-dihydroxyphenylalanine in rat treated neonatally with 6-hydroxydopamine.J. Pharm. Exp. Ther. 235, 287–295.

3. Breese GR, TC Napier and RA Mueller (1985b) Dopamine agonist-induced locomotor activity in rats treated with 6-hydroxydopamine at differing ages: Functional supersensitivity of D-1 dopamine receptors in neonatally-lesioned rats.J. Pharm. Exp. Ther. 234, 447–455.

4. Breese GR, GE Duncan, TC Napier, SC Bondy, LC Iorio and RA Mueller (1987) 6-Hydroxydopamine treatment enhance behavioral responses to intracerebral microinjection of D(1)- and D(2)-dopamine agonists into nucleus accumbens and striatum without changing dopamine antagonist binding.J. Pharm. Exp. Ther. 240, 167–176.

5. Briggs CA, NJ Pollock, DE Frail, CL Paxson, RF Rakowski, CH Kang and JW Kebabian (1991) Activation of the 5-HT1C receptor expressed inXenopus oocytes by the benzazepines SCH 23390 and SKF 38393.Br. J. Pharmacol. 104, 1038–1044.

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