Arrestin Regulation of Small GTPases
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Publisher
Springer Berlin Heidelberg
Link
http://link.springer.com/content/pdf/10.1007/978-3-642-41199-1_19
Reference23 articles.
1. Anthony DF, Sin YY et al (2011) beta-Arrestin 1 inhibits the GTPase-activating protein function of ARHGAP21, promoting activation of RhoA following angiotensin II type 1A receptor stimulation. Mol Cell Biol 31(5):1066–1075
2. Aoki H, Izumo S et al (1998) Angiotensin II activates RhoA in cardiac myocytes: a critical role of RhoA in angiotensin II-induced premyofibril formation. Circ Res 82(6):666–676
3. Barnes WG, Reiter E et al (2005) beta-Arrestin 1 and Galphaq/11 coordinately activate RhoA and stress fiber formation following receptor stimulation. J Biol Chem 280(9):8041–8050
4. Berthouze-Duquesnes M, Lucas A et al (2013) Specific interactions between Epac1, beta-arrestin2 and PDE4D5 regulate beta-adrenergic receptor subtype differential effects on cardiac hypertrophic signaling. Cell Signal 25(4):970–980
5. Bhattacharya M, Anborgh PH et al (2002) Beta-arrestins regulate a Ral-GDS Ral effector pathway that mediates cytoskeletal reorganization. Nat Cell Biol 4(8):547–555
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1. β-Arrestins: Multitask Scaffolds Orchestrating the Where and When in Cell Signalling;Beta-Arrestins;2019
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