The Role of Mutations in the Deoxycytidine Kinase (dCK) Gene in the Development of Acute Myeloid Leukemia (AML) Towards Relapsed and/or Refractory Disease

Author:

Van den Heuvel-Eibrink M. M.,Wiemer E. A. C.,Kuipers M.,Pieters R.,Sonneveld P.

Publisher

Springer Berlin Heidelberg

Reference29 articles.

1. Bennett JMD, Catovsky D, et al (1985) Proposed revised criteria for the classification of acute myeloid leukemia. A report of the French-American-British Cooperative Group. Ann Intern Med 103:620–625

2. Bhalla K, Nayak R, et al (1984) Isolation and characterization of a deoxycytidine kinase-deficient human promyelocytic leukemic cell line highly resistant to 1-beta-D-arabinofuranosylcytosine. Cancer Res 44:5029–37

3. Chottiner EG, Shewach DS, et al (1991) Cloning and expression of human deoxycytidine kinase cDNA. Proc Natl Acad Sci U S A 88:1531–5

4. Colly LP, Peters WG, et al (1987) Deoxycytidine kinase and deoxycytidine deaminase values correspond closely to clinical response to cytosine arabinoside remission induction therapy in patients with acute myelogenous leukemia. Semin Oncol 14:257–61

5. Dow LW Dahl GV, et al (1986) Correlation of drug sensitivity in vitro with clinical responses in childhood acute myeloid leukemia. Blood 68 (2):400–5

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