ABT-737 suppresses aberrant Hedgehog pathway and overcomes resistance to smoothened antagonists by blocking Gli
Author:
Funder
Chinese Natural Science Foundation
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology,Hematology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s12032-022-01794-w.pdf
Reference45 articles.
1. Jiang J, Hui CC. Hedgehog signaling in development and cancer. Dev Cell. 2008;15(6):801–12. https://doi.org/10.1016/j.devcel.2008.11.010.
2. Wu F, et al. Hedgehog signaling: From basic biology to cancer therapy. Cell Chem Biol. 2017;24(3):252–80. https://doi.org/10.1016/j.chembiol.2017.02.010.
3. Raleigh DR, Reiter JF. Misactivation of Hedgehog signaling causes inherited and sporadic cancers. J Clin Invest. 2019;129(2):465–75. https://doi.org/10.1172/jci120850.
4. Reifenberger J, et al. Missense mutations in SMOH in sporadic basal cell carcinomas of the skin and primitive neuroectodermal tumors of the central nervous system. Cancer Res. 1998;58(9):1798–803.
5. Pugh TJ, et al. Medulloblastoma exome sequencing uncovers subtype-specific somatic mutations. Nature. 2012;488(7409):106–10. https://doi.org/10.1038/nature11329.
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