Influence of prostaglandin-synthesis inhibitors on carbachol-and histamine-induced vasodilatation in perfused rat hindquarters
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/BF00580263.pdf
Reference18 articles.
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3. Casteels R, Suzuki H (1980) The effect of histamine on the smooth muscle cells of the ear artery of the rabbit. Pfl�gers Arch 387:17?25
4. Chand N, Altura BM (1981a) Acetylcholine and bradykinin relax intrapulmonary arteries by acting on endothelial cells: role in lung vascular diseases. Science 213:1376?1379
5. Chand N, Altura BM (1981b) Lipoxygenases mediate endothelial celldependent relaxations to acetylcholine and bradykinin in canine renal arteries. Circulation 64:suppl IV:284.
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1. NO release and the opening of K+ATP channels mediate vasodilator responses to histamine in the cat;American Journal of Physiology-Heart and Circulatory Physiology;1997-08-01
2. Renal vasodilation to histaminein vitro: Roles of nitric oxide, cyclo-oxygenase products and H2 receptors;Inflammation Research;1995-03
3. Differential Effects of Agents on Bronchial and Vascular Tone and Lung Weight in the Rat Isolated Perfused Lung;Pulmonary Pharmacology;1994-08
4. Histamine-induced venodilation in human beings involves both H1 and H2 receptor subtypes;Journal of Allergy and Clinical Immunology;1994-03
5. Histamine elicits competing endothelium-dependent constriction and endothelium-independent dilation in vivo in mouse cerebral arterioles.;Stroke;1990-02
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