Author:
Mayer R. John,Landon Michael,Lowe James
Reference203 articles.
1. Agundez, J. A., Jimenez, F., Luengo, A., Bernai, M. L., Molina, J. A., Ayuso, L., Vazquez, A., Parra, J., Duarte, J., Coria, F., and Ladero, J. M., 1995, Association between the oxidative polymorphism and early onset of Parkinson’s disease, Clin. Pharmacol. Ther.
57:291–298.
2. Ahrens, P. B., Besancon, F., Memet, S., and Ankel, H., 1990, Tumour necrosis factor enhances induction by beta-interferon of a ubiquitin cross-reactive protein, J. Gen. Virol.
71:1675–1682.
3. Albrecht, S., and Bilbao, J. M., 1993, Ubiquitin expression in inclusion body myositis. An immuno-histochemical study, Arch. Pathol. Lab. Med.
117:789–793.
4. Arnold, J. E., Tipler, C., Laszlo, L., Hope, J., Landon, M., and Mayer, R. J., 1995, The abnormal isoform of the prion protein accumulates in late-endosome-like organelles in scrapie-infected mouse brain, J. Pathol.
176:403–411.
5. Askanas, V., and Engel, W. K., 1995, New advances in the understanding of sporadic inclusion-body myositis and hereditary inclusion-body myopathies, Curr. Opin. Rheumatol.
7:486–496.
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