Hypothalamic-Pituitary Function during Alcohol Exposure and Withdrawal and Cocaine Exposure-Reference-Cited by-同舟云学术

Hypothalamic-Pituitary Function during Alcohol Exposure and Withdrawal and Cocaine Exposure

Published:1992 Issue: Volume: Page:57-71
ISSN:0738-422X
Container-title:Recent Developments in Alcoholism
language:
Short-container-title:

Author:

Wilkins Jeffery N.,Gorelick David A.,Nademanee Koonlawee,Taylor Anna,Herzberg David S.

Publisher

Springer US

Link

http://link.springer.com/content/pdf/10.1007/978-1-4899-1648-8_3

Reference39 articles.

1. Gorelick DA, Wilkins JN: Special aspects of human alcohol withdrawal, in Galanter M (ed): Recent Developments in Alcoholism, Vol. IV New York, Plenum Press, 1986, p 285-1285.

2. Loosen PT, Dew BW, Prange AJ: Long-term predictors of outcome in abstinent alcoholic men. Am J Psychiatry 147:1662–1666, 1990.

3. Teoh SK, Mendelson JH, Mello NK, Weiss R, McElroy S, McAfee B: Hyperprolactinemia and risk for relapse of cocaine abuse. Biol Psychiatry 28:824–828, 1990.

4. Mendelson JH, Teoh SK, Lange U, Mello NK, Weiss R, Skupny A, Ellingboe J: Anterior pituitary, adrenal, and gonadal hormones during cocaine withdrawal. Am J Psychiatry 145(9):1094–1098, 1988.

5. Wilkins JN, Gorelick DA, Bamshad BR, Setoda DY: Elevated serum prolactin levels in newly abstinent male cocaine abusers. Society for Neuroscience, Toronto, Canada, November 1988 (abstract).

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