Mouse Models of Human Trisomy 21 (Down Syndrome)
Author:
Publisher
Birkhäuser Boston
Link
http://link.springer.com/content/pdf/10.1007/978-1-4899-6732-9_1
Reference111 articles.
1. Ault B, Caviedes P, Rapoport SI (1989): Neurophysiological abnormalities in cultured dorsal root ganglion neurons from the trisomy 16 mouse fetus, a model for Down syndrome. Brain Res 485:165–170.
2. Avraham KB, Schickler M, Groner Y (1988): Down’s syndrome: abnormal neuromuscular junction in tongue of transgenic mice with elevated levels of human Cu/Zn-Superoxide dismutase. Cell 54:823–829.
3. Bacchus C, Sterz H, Buselmaier W, Sahai S, Winking H (1987): Genesis and systematization of cardiovascular anomalies and analysis of skeletal malformations in murine trisomy 16 and 19. Hum Genet 77:12–22.
4. Balazs R, Brooksbank BWL (1985): Neurochemical approaches to pathogenesis of Down’s syndrome. J Ment Defic Res 29:1–14.
5. Bendotti C, Forloni GL, Morgan RA, O’Hara BF, Oster-Granite ML, Reeves RH, Gearhart JD, Coyle JT (1988): Neuroanatomical localization and quantification of amyloid precursor protein mRNA by in situ hybridization in the brains of normal, aneuploid, and lesioned mice. Proc Natl Acad Sci USA 85:3628–3632.
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