Advanced glycation end products promote proliferation of cardiac fibroblasts by upregulation of KCa3.1 channels-Reference-Cited by-同舟云学术

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Advanced glycation end products promote proliferation of cardiac fibroblasts by upregulation of KCa3.1 channels

Published:2012-10-02 Issue:6 Volume:464 Page:613-621
ISSN:0031-6768
Container-title:Pflügers Archiv - European Journal of Physiology
language:en
Short-container-title:Pflugers Arch - Eur J Physiol

Author:

Zhao Li-Mei,Zhang Wei,Wang Li-Ping,Li Gui-Rong,Deng Xiu-Ling

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00424-012-1165-0.pdf

Reference29 articles.

1. Aronson D (2003) Cross-linking of glycated collagen in the pathogenesis of arterial and myocardial stiffening of aging and diabetes. J Hypertens 21:3–12

2. Asbun J, Villarreal FJ (2006) The pathogenesis of myocardial fibrosis in the setting of diabetic cardiomyopathy. J Am Coll Cardiol 47:693–700

3. Avendano G, Dharamsey S, Dasmahapatra A, Agarwal R, Reddi A, Regan T (1996) Left ventricular diastolic function in hypertension and role of plasma glucose and insulin: comparison with diabetic heart. Circulation 93:1396–1402

4. Berg TJ, Snorgaard O, Faber J, Torjesen PA, Hildebrandt P, Mehlsen J, Hanssen KF (1999) Serum levels of advanced glycation end products are associated with left ventricular diastolic function in patients with type 1 diabetes. Diabetes Care 22:1186–1190

5. Candido R, Forbes JM, Thomas MC, Thallas V, Dean RG, Burns WC, Tikellis C, Ritchie RH, Twigg SM, Cooper ME, Burrell LM (2003) A breaker of advanced glycation end products attenuates diabetes-induced myocardial structural changes. Circ Res 92:785–792

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