Kv1.5 blockers preferentially inhibit TASK-1 channels: TASK-1 as a target against atrial fibrillation and obstructive sleep apnea?-Reference-Cited by-同舟云学术

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Kv1.5 blockers preferentially inhibit TASK-1 channels: TASK-1 as a target against atrial fibrillation and obstructive sleep apnea?

Published:2014-12-17 Issue:5 Volume:467 Page:1081-1090
ISSN:0031-6768
Container-title:Pflügers Archiv - European Journal of Physiology
language:en
Short-container-title:Pflugers Arch - Eur J Physiol

Author:

Kiper Aytug K.,Rinné Susanne,Rolfes Caroline,Ramírez David,Seebohm Guiscard,Netter Michael F.,González Wendy,Decher Niels

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00424-014-1665-1.pdf

Reference38 articles.

1. Bachmann A, Gutcher I, Kopp K, Brendel J, Bosch RF, Busch AE, Gogelein H (2001) Characterization of a novel Kv1.5 channel blocker in Xenopus oocytes, CHO cells, human and rat cardiomyocytes. Naunyn Schmiedebergs Arch Pharmacol 364:472–478

2. Bayliss DA, Sirois JE, Talley EM (2003) The TASK family: two-pore domain background K+ channels. Mol Interv 3:205–219

3. Burashnikov A, Di Diego JM, Zygmunt AC, Belardinelli L, Antzelevitch C (2007) Atrium-selective sodium channel block as a strategy for suppression of atrial fibrillation: differences in sodium channel inactivation between atria and ventricles and the role of ranolazine. Circulation 116:1449–1457

4. Burashnikov A, Barajas-Martinez H, Hu D, Nof E, Blazek J, Antzelevitch C (2012) Atrial-selective prolongation of refractory period with AVE0118 is due principally to inhibition of sodium channel activity. J Cardiovasc Pharmacol 59:539–546

5. Campagna-Slater V, Arrowsmith AG, Zhao Y, Schapira M (2010) Pharmacophore screening of the protein data bank for specific binding site chemistry. J Chem Inf Model 50:358–367

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3. The Contribution of Genetic Sequencing Information to the Identification and Functional Characterization of Two-Pore Domain Potassium (K2P) Channels as Viable Therapeutic Targets;Ion Channels as Targets in Drug Discovery;2024

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