SNAT2 is responsible for hyperosmotic induced sarcosine and glycine uptake in human prostate PC-3 cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s00424-022-02752-1.pdf
Reference41 articles.
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2. Anderson CM, Jevons M, Thangaraju M, Edwards N, Conlon NJ, Woods S, Ganapathy V, Thwaites DT (2010) Transport of the photodynamic therapy agent 5-aminolevulinic acid by distinct H+-coupled nutrient carriers coexpressed in the small intestine. J Pharmacol Exp Ther 332:220–228. https://doi.org/10.1124/jpet.109.159822
3. Atkinson BN, Bell SC, De Vivo M, Kowalski LR, Lechner SM, Ognyanov VI, Tham CS, Tsai C, Jia J, Ashton D, Klitenick MA (2001) ALX 5407: a potent, selective inhibitor of the hGlyT1 glycine transporter. Mol Pharmacol 60:1414–1420. https://doi.org/10.1124/mol.60.6.1414
4. Baum CE, Price DK, Figg WD (2010) Sarcosine as a potential prostate cancer biomarker and therapeutic target. Cancer Biol Ther 9:341–342. https://doi.org/10.4161/cbt.9.5.11310
5. Bevilacqua E, Bussolati O, Dall’Asta V, Gaccioli F, Sala R, Gazzola GC, Franchi-Gazzola R (2005) SNAT2 silencing prevents the osmotic induction of transport system A and hinders cell recovery from hypertonic stress. FEBS Lett 579:3376–3380. https://doi.org/10.1016/j.febslet.2005.05.002
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