Visfatin induces oxidative stress in differentiated C2C12 myotubes in an Akt- and MAPK-independent, NFĸB-dependent manner-Reference-Cited by-同舟云学术

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Visfatin induces oxidative stress in differentiated C2C12 myotubes in an Akt- and MAPK-independent, NFĸB-dependent manner

Published:2009-11-08 Issue:4 Volume:459 Page:619-630
ISSN:0031-6768
Container-title:Pflügers Archiv - European Journal of Physiology
language:en
Short-container-title:Pflugers Arch - Eur J Physiol

Author:

Oita Radu C.,Ferdinando Dudley,Wilson Steve,Bunce Christopher,Mazzatti Dawn J.

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00424-009-0752-1.pdf

Reference59 articles.

1. Aam BB, Myhre O, Fonnum F (2003) Transcellular signalling pathways and TNF-alpha release involved in formation of reactive oxygen species in rat alveolar macrophages exposed to tert-butylcyclohexane. Arch Toxicol 77(12):678–684

2. Adams JD Jr (2008) Alzheimer's disease, ceramide, visfatin and NAD. CNS Neurol Disord Drug Targets 7(6):492–498

3. Adya R, Tan BK, Punn A et al (2008) Visfatin induces human endothelial VEGF and MMP-2/9 production via MAPK and PI3K/Akt signalling pathways: novel insights into visfatin-induced angiogenesis. Cardiovasc Res 78(2):356–365

4. Aldhahi W, Hamdy O (2003) Adipokines, inflammation, and the endothelium in diabetes. Curr Diab Rep 3(4):293–298

5. Anrather J, Racchumi G, Iadecola C (2006) NF-kappaB regulates phagocytic NADPH oxidase by inducing the expression of gp91phox. J Biol Chem 281(9):5657–5667

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