How kidney function adapts to changes in acid–base balance: the AE4 transporter as a central acid–base sensor in tubular cells
Author:
Funder
Universität Regensburg
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s00424-023-02898-6.pdf
Reference5 articles.
1. Chambrey R, Kurth I, Peti-Peterdi J, Houillier P, Purkerson JM, Leviel F, Hentschke M, Zdebik AA, Schwartz GJ, Hübner CA, Eladari D (2013) Renal intercalated cells are rather energized by a proton than a sodium pump. Proc Natl Acad Sci U S A 110(19):7928–7933. https://doi.org/10.1073/pnas.1221496110
2. Kurtz I (2018) Renal tubular acidosis: H+/base and ammonia transport abnormalities and clinical syndromes. Adv Chronic Kidney Dis 25(4):334–350. https://doi.org/10.1053/j.ackd.2018.05.005
3. Vitzthum H, Koch M, Eckermann L, Svendsen SL, Berg P, Hübner CA, Wagner CA, Leipziger J, Meyer-Schwesinger C, Ehmke H (2023) The AE4 transporter mediates kidney acid-base sensing. Nat Commun 14(1):3051. https://doi.org/10.1038/s41467-023-38562-x
4. Wagner CA, Imenez Silva PH, Bourgeois S (2019) Molecular pathophysiology of acid-base disorders. Semin Nephrol 39(4):340–352. https://doi.org/10.1016/j.semnephrol.2019.04.004
5. Wall SM, Verlander JW, Romero CA (2020) The renal physiology of pendrin-positive intercalated cells. Physiol Rev 100(3):1119–1147. https://doi.org/10.1152/physrev.00011.2019
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