Neuronal Cav3 channelopathies: recent progress and perspectives
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s00424-020-02429-7.pdf
Reference172 articles.
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2. Andrade A, Hope J, Allen A, Yorgan V, Lipscombe D, Pan JQ (2016) A rare schizophrenia risk variant of CACNA1I disrupts CaV3.3 channel activity. Sci Rep 6:34233. https://doi.org/10.1038/srep34233
3. Aromolaran KA, Benzow KA, Cribbs LL, Koob MD, Piedras-Renteria ES (2010) T-type current modulation by the actin-binding protein Kelch-like 1. Am J Physiol Cell Physiol 298:C1353–C1362. https://doi.org/10.1152/ajpcell.00235.2009
4. Asmara H, Micu I, Rizwan AP, Sahu G, Simms BA, Zhang FX, Engbers JDT, Stys PK, Zamponi GW, Turner RW (2017) A T-type channel-calmodulin complex triggers alphaCaMKII activation. Mol Brain 10:37. https://doi.org/10.1186/s13041-017-0317-8
5. Astori S, Wimmer RD, Prosser HM, Corti C, Corsi M, Liaudet N, Volterra A, Franken P, Adelman JP, Luthi A (2011) The Ca(V)3.3 calcium channel is the major sleep spindle pacemaker in thalamus. Proc Natl Acad Sci U S A 108:13823–13828. https://doi.org/10.1073/pnas.1105115108
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