Late sodium current associated cardiac electrophysiological and mechanical dysfunction-Reference-Cited by-同舟云学术

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Late sodium current associated cardiac electrophysiological and mechanical dysfunction

Published:2017-11-10 Issue:3 Volume:470 Page:461-469
ISSN:0031-6768
Container-title:Pflügers Archiv - European Journal of Physiology
language:en
Short-container-title:Pflugers Arch - Eur J Physiol

Author:

Yu Shandong,Li Gang,Huang Christopher L.-H.,Lei Ming,Wu Lin

Funder

Chinese National Natural Sciences Foundation

Chinese National Natural Science Foundation

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Link

http://link.springer.com/article/10.1007/s00424-017-2079-7/fulltext.html

Reference103 articles.

1. Abriel H (2010) Cardiac sodium channel Na(v)1.5 and interacting proteins: physiology and pathophysiology. J Mol Cell Cardiol 48:2–11. https://doi.org/10.1016/j.yjmcc.2009.08.025

2. Ahern GP, Hsu SF, Klyachko VA, Jackson MB (2000) Induction of persistent sodium current by exogenous and endogenous nitric oxide. J Biol Chem 275:28810–28815. https://doi.org/10.1074/jbc.M003090200

3. Akalin F, Tirtir A, Yilmaz Y (2003) Increased QT dispersion in epileptic children. Acta Paediatr 92:916–920

4. Aldakkak M, Camara AK, Heisner JS, Yang M, Stowe DF (2011) Ranolazine reduces Ca2+ overload and oxidative stress and improves mitochondrial integrity to protect against ischemia reperfusion injury in isolated hearts. Pharmacol Res 64:381–392. https://doi.org/10.1016/j.phrs.2011.06.018

5. Antzelevitch C (2000) Electrical heterogeneity, cardiac arrhythmias, and the sodium channel. Circ Res 87:964–965

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